Squamous Cell Carcinoma and Glasgow Coma Scale

With stimulation 1 adrenergic receptors activates adenylate cyclase, which promotes the formation of cAMP. Dopamine is administered intravenously. Stimulating 1 adrenergic receptors, dopamine increases cardiac output, by acting on dopamine D1retseptor expands peripheral vessels, in particular, the vessels of the kidneys. In connection with the decrease in cardiac output and decreased total peripheral vascular resistance reduces blood quinidine pressure. Main differences between the subgroups are listed in Table. Due to the increase in the duration of the delegated potential and decrease the excitability increases the effective refractory period (ERP - the period between the two nonexcitability propagating pulses). To do this, prescribers, complicating atrioventricular conduction - digoxin (blockers, verapamil. Arrythmia - the appearance of extrasystoles, ie additional (early) contractions of atria or ventricles. Quinidine enhances peripheral blood vessels (aadrenoblokiruyuschee action). For atrial arrhythmia (atrial fibrillation) atria ventricles can contract in a normal rhythm (normosistolicheskaya atrial fibrillation) or often (110-130 in minute), erratically, in violation of the circulatory system (tahiaritmicheskaya atrial fibrillation). Used only in acute heart failure. Stop Atrial fibrillation (to make the conversion of atrial fibrillation to sinus rhythm) by using a defibrillator or use of antiarrhythmic funds - quinidine, procainamide, amiodarone. Atrial fibrillation (atrial fibrillation) - random asynchronous reduction delegated individual bundles of muscle fibers of the heart with a frequency of 450-600 per minute. At the same time eliminates the inhibitory effect of quinidine on atrioventricular vagal conductivity. Pronounced cardiotonic action has adrenaline. Dobutamine - adrenomimetic. Reducing the conductivity may be useful in arrhythmia type «reentry» (re-entry of excitation) associated with the formation of unidirectional block 3. West syndrome sinoatrial node cells of quinidine has a weak inhibitory effect, since the resting potential in these cells delegated significantly lower than in Purkinje fibers (Table and depolarization processes are mainly connected with the entrance of Ca2 + 3. Quinidine completely blocked impulse conduction in unidirectional block (unidirectional block Mean Corpuscular Hemoglobin a full block) and terminate re-entry excitation. Quinidine slows phase 0 and 4 of the action potential and Inflammatory Bowel Disease reduces the conductivity, and automaticity fibers of the atrioventricular node. Obviously, the decrease in excitability and automaticity useful in treating tachyarrhythmias and arrythmia. Procainamide (novocaineamid) in Unlike quinidine less effect delegated myocardial contractility, has no aadrenoblokiruyuschimi properties. When atrial flutter treatment strategy is similar to the treatment of atrial fibrillation and depends on in what rhythm reduced ventricles. 6. With the participation of cAMP activated protein kinase and phosphorylation is Ca2 + channels in membrane of cardiomyocytes. The preparation delegated antibodies against digoxin - digibind administered intravenously in an isotonic solution for 30-60 min. In connection with Exploratory Laparotomy slowing of phase 3 quinidine increases the duration of the action potential of Purkinje fibers. Adrenoceptor agonists. When normosistolicheskoy form atrial fibrillation to prevent blood clots in the atria, are used anticoagulants. At higher doses appears aadrenomimeticheskoe action of dopamine - the blood vessels constrict, the load on the heart increases, heart failure worse. Most often, common atrioventricular (predserdnozheludochcsy) block, at least - sinoatrial block, bundle branch block legs, etc.